Preliminary experiments point out conolidine could inhibit certain ion channels, decreasing neuronal excitability and restricting suffering alerts. This system is especially appropriate in neuropathic ache, in which irregular signaling causes persistent pain. Also, conolidine appears to influence G protein-coupled receptor (GPCR) pathways integral to suffering notion. Computational do... https://horacex691zxr0.bloguerosa.com/profile